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Phenergan 25 mg 50 tablets ) was used for treatment. In patients taking antipsychotics, the dosage of chlorpromazine was reduced to 2 mg achieve an elimination half-life of 20 to 30 hours. In patients taking anticonvulsant drugs, chlorpromazine dose was reduced to 1 mg per day achieve an elimination half-life of 6 to 8 h. Study 1. The primary hypothesis was that long-term use of chlorpromazine would result in a reduction cognitive function and that this reduction would be more pronounced in patients with Alzheimer's Disease. The secondary hypothesis was that chlorpromazine would reduce the level of plasma phosphatidylcholine (PCH). Methods Study 1 Adult patients with mild to moderate Alzheimer Disease were randomized either to receive chlorpromazine 25 mg or placebo, in a 1:1 ratio, every day, for six months. Patients were also randomized to receive either 40 mg of fluoxetine or placebo, in a 1:1 ratio, every day, for six months. The study was approved by institutional review board of the University Illinois at Chicago. Treatment Chlorpromazine 25 mg, 40 and placebo were given as daily doses, either by oral or intravenous ingestion. Patients were asked to continue taking their usual antipsychotic and antihypertensive medications during the study. Primary Outcome The primary outcome was percent change compared with baseline in the total score on Mini-Mental State Examination, which was performed at baseline and six months (t = 3.5) and at the end of study (t = 6.5). The Mini-Mental State Examination (MMSE) is a standard test of cognitive performance. The scoring system is as follows: 1 = normal range of functioning; 10 = severe dementia; 20 memory dysfunction; 40 = total dementia (clinical diagnosis or by a cognitively trained neurologist). Secondary Outcomes The secondary outcomes were change in the cognitive functions tested at each time point, the percentage change in plasma levels of phosphatidylcholine (PCH), the number cognitive lapses, and of abnormal tests on the MMSE. total score MMSE was analyzed as a total of 24 cognitive can i get phenergan over the counter functions in order to detect a significant change in cognitive function. In order to test the hypothesis that chlorpromazine would reduce cognitive function, the following functions were assessed at each time point: (a) Number of Cognitive Lapses: The lapses was determined by scoring system of the Mini-Mental State Examination (MMSE). scoring system is as follows: 1 = normal range of functioning; 10 = severe dementia; 20 memory dysfunction; 40 = total dementia (clinical diagnosis or by a cognitively trained neurologist). (b) Number of Nonfunctional Tests: Each cognitive function was compared to the threshold of total score. number nonfunctional tests was determined by the scoring system of Mini-Mental State Examination (MMSE). The scoring system is as follows: 1 = normal range of functioning; 10 = severe dementia; 20 memory dysfunction; 40 = total dementia (clinical diagnosis or by a cognitively trained neurologist). (c) Percentage Change in PCH and Inhibition of the Receptor For Chlorpromazine at Serum: The percentage change in plasma levels of PCH and the inhibition receptor for chlorpromazine at serum were determined as described in the Supplementary Appendix, available with full text of this article at NEJM.org. Results At six months, both chlorpromazine 25 mg and fluoxetine 40 were can i buy phenergan over the counter in uk associated with a greater reduction drugstore online discount code in MMSE score compared with placebo (P <.05) (Figure 1). In the placebo group, MMSE score declined by 1.5 points at six months, whereas there was a trend toward greater reduction in the chlorpromazine group (P =.09). The percentage of cognitive lapses was also lower in the chlorpromazine group (45% compared with 55% at six months) (P <.05). In the clinical trial population, there was a greater reduction in the number of cognitive lapses in the chlorpromazine group at six months than in the fluoxetine group (P =.05). There was a trend toward greater reduction in cognitive lapses the chlorpromazine group at six months compared with the fluoxetine group (P =.10) (Figure 2). Patients with Alzheimer's Disease were less likely to have cognitive lapses or a significant decrease in cognitive function the chlorpromazine group than in fluoxetine (P <.05) (Figure 3). In the clinical trial population, there was a greater reduction in total neurocognitive function the chlorpromazine group at six.



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